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{ <http://www.tkuhn.ch/bel2nanopub/RAcMAhXyEY8hnCeglApRI1Cb5nJcWrEMwESNpNlUDWko8#_5> ?p ?o ?g. }
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We have found that the ER chaperone BiP/GRP78 binds ATF6 and dissociates in response to ER stress. Loss of BiP binding correlates with the translocation of ATF6 to the Golgi, which was slowed in cells overexpressing BiP.
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