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- _4 value "Acute lung injury induced by intrapulmonary deposition of IgG immune complexes in rats exhibits a pathophysiology similar to that observed during sepsis, ischemia/ reperfusion, or trauma.12?15 In this model, enhanced production of tumor necrosis factor-a (TNF-a) and interleukin-1b (IL-1b) by activated lung macrophages causes up-regulation of the adhesion molecules, intercellular adhesion molecule-1 (ICAM-1), and E-selectin on the pulmonary endothelium.16,17" provenance.
- _4 wasQuotedFrom 9916938 provenance.