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{ <http://www.tkuhn.ch/bel2nanopub/RAk92ALGaUVWg7TVtyNDaWowSBVUxwO4MENa49CBbbnMk#_3> ?p ?o ?g. }
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Previously, it was shown that proinflammatory cytokines characteristic of the APR (TNF-alpha, IL-1beta, and IFNgamma) induced the expression of the PRL receptor (PRLR) by pulmonary fibroblasts in vitro.
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