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{ <http://www.tkuhn.ch/bel2nanopub/RAkCXneVFeSUuRpD6Nzv3dCv8I0wiA8T3C12V_GS7Ja_g#_2> ?p ?o ?g. }
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These results implicate NO and prostacyclin produced by the interaction of VEGF/VPF with its Flk-1/KDR/VEGF-R2 receptor as mediators of VEGF/VPF-induced vascular permeability.
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