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{ <http://www.tkuhn.ch/bel2nanopub/RAlpOcLs_N2JQZ_10_Qj-iD46D24NuyiAmHZMhLaVLSMA#_5> ?p ?o ?g. }
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Simultaneous treatment with OA plus dibutyryl cAMP caused a synergistic up-regulation of steady-state levels of HO-1 mRNA, and the specific protein kinase A inhibitor KT5720 markedly reduced the OA-dependent HO-1 mRNA induction.
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