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{ <http://www.tkuhn.ch/bel2nanopub/RAmJokjGq0CANPTYaL4xVAou2ARJuxUiJZePl5LFT_izI#_3> ?p ?o ?g. }
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Methylprednisolone (MP), a synthetic glucocorticoid, partially inhibited the injury-induced expression of TNFR1 and TNFR2, an effect which could be reversed by RU486, an antagonist of glucocorticoid receptors.
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