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{ <http://www.tkuhn.ch/bel2nanopub/RAmR192VqsZ6T4m088V6vondwQ95fuoDn36wGiM8wAbvU#_4> ?p ?o ?g. }
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Ca(2+) influx was thought to trigger FGF-1 synthesis. Such Ca(2+)-mediated enhancement of FGF-1 synthesis, however, did not occur in the presence of norepinephrine (NE), but was restored by KT-5720, an inhibitor of protein kinase A (PKA)
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