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{ <http://www.tkuhn.ch/bel2nanopub/RAnazDmcnzFS4s_OR_FNaWbYNEU2zxmWLzJNsTa-EJ-sk#_2> ?p ?o ?g. }
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Here we show that conditional deletion of Pot1a elicits a DNA damage response at telomeres, resulting in p53-dependent replicative senescence.
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