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{ <http://www.tkuhn.ch/bel2nanopub/RAtMFXFMz1w-uE2I6eEwjTTgZK8JBSCyxguk1n9mWm6n4#_2> ?p ?o ?g. }
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These results indicate that the increase in mitochondrial reactive oxygen species from knockout of SOD2 activity can result in biochemical aberrations with features reminiscent of mitochondrial myopathy
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