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{ <http://www.tkuhn.ch/bel2nanopub/RAtUCGAUpQwWpFyOO9N3lVNMb6gM7566KRdEF2C3VdTZo#_5> ?p ?o ?g. }
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The interaction between PP5 and ASK1 was induced by H(2)O(2) treatment and was followed by the decrease in ASK1 activity. PP5 inhibited not only H(2)O(2)-induced sustained activation of ASK1 but also ASK1-dependent apoptosis
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