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{ <http://www.tkuhn.ch/bel2nanopub/RAyq1FeVmGBq6xcB5QX9u_FrfSIMCHudYqmxD2m9-W4kk#_4> ?p ?o ?g. }
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Compared to wild-type mice, IL-12Rbeta2-deficient mice exhibited significantly increased autoantigen-induced proliferative response and increased production of TNF-alpha, GM-CSF, IL-17, IL-18/IL-18Ralpha, and NO.
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