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{ <http://www.tkuhn.ch/bel2nanopub/RAz5fA-OoW_7Mb05H2HSvX78TNavj92HSkeWBN-vFy5GA#_6> ?p ?o ?g. }
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Pharmacologic inhibition of FAK or expression of kinase-deficient FAK, mutated by substituting Tyr-397 with Phe, inhibit TGF-beta1-induced alpha-smooth muscle actin expression, stress fiber formation, and cellular hypertrophy.
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_6
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12531888
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